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Diabetes Type 2 Obesity Prediabetes DiObesity Weight Loss Diabetes Metabolic Appetite Diabetes Type 2  - Obesity Prediabetes DiObesity Weight Loss Diabetes Metabolic Appetite
Updated Jan 4th 2015

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Obesity Prediabetes DiObesity Diabetes Recovery Protocols >>
 
CORE PROTOCOL >>


DiabetaLean two caps twice per day
 
Minerals PLUS one cap twice per day
 
Leptin Metabolic one cap in AM
 
Carbo Block two caps twice before all meals and snacks
 
GlycemX one to meals and snacks as smoothies twice per day

BerberStatin two to three capsules twice per day

Carnosine two capsules twice per day

NutriTRALA one tablet three times per day

VitaMineralMAX one to two capsules before each meal or snack

Minerals PLUS one capsule twice per day


Chromium Cruciferate one capsule before meals three times per day

MyCO D2 three capsules twice per day

Nutriodine gradually increase from one to five drops up to 15 drops topically on arms or in filtered water three times per day

Carnosine two capsules three times per day


ADVANCED PROTOCOLS >>

Mood Balance BH4 Biopterin one capsule twice per day

Norival one capsule twice per day

Hoodia 2000 TR Two tabs AM and middle of afternoon

Green Tea Supreme one capsule three times per day

MitoThyroid two capsules AM and middle of the afternoon

NutriDine 5 drops three times per day topically or in filtered non-chlorinated or fluoridated distilled or Pure Water Systems water

IndiumEase 5 to 6 drops under tongue AM and bedtimes on an empty stomach with no food or drink except water for twenty minutes

COMPLETE  PROTOCOLS >> 

VibraSlim 10 minutes static resistance exercises once to twice per day

CALMind one to two scoops four times per day

Super Acai 1200 one capsule three times per day

CoQ10 Supreme one softgels two to three times per day

LipoFlush two softgels two to three times per day


COMPLETE PROTOCOL >>


Mitochondrial Catalysts one capsule three times per day

Mitocarnitine one capsule three times per day

SuperNOX one tab or VascuNOX one tspn in water three times per day

AntiAgeMAX one capsule three times per day


Meal Replacements with GlycemX Mint Chocolate or Vanilla one to two meals and snacks per day - 2 oz of Almond-Coconut Mild 6 oz filtered water in Bullet Blender or equivalant

OmegaSupreme or Omega 3 6 9 one softgels three times per day OR... Omega 3 6 9 Emulsion one tspn three times per day in diluted orange juice 1 oz in 4 oz of water
 
LOWER Carbohydrate Lower Glycemic Index Diet - Maximum 1200 calories per day in six small meals
 

VibraSlim 10 minutes dynamic stretching exercise per day

Walking Program 30 minutes per day 

LUMEN PHOTON, MEDITHERA, PMT100 ENERGETIC TECHNOLOGIES & INFRARED SAUNA TO RAISE ADIPOSE FAT MOBILIZATION FROM LIPOTOMES FROM SKIN AND CENTRAL ABDOMINAL CAVITY ORGANS

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We should all be familiar with the idea that food intake is energy intake. And when one eats too much, they are taking in too much energy - and if this energy is not used in active processes in the body, it will be stored. Most of it will be stored as fat. Obese states occur when energy intake chronically exceeds energy expenditure, as all the excess energy is locked up in the extensive fat reserves. The specialised cells which store fat are called adipocytes, and fatty tissue is referred to as adipose. An interesting observation is that an adult who becomes obese has the same number of adipocytes as they did when they were lean. The idea being that at around the age of 8 or so, one stops producing any significant number of adipocytes. Past this, more fat is packed into the existing cells, so they just swell up. It seems that some people may be primed to put on more weight because of a greater existing number of adipocytes produced during childhood, because they have more ‘storage space’ in which to pack fat. light micrograph of adipose tissue Adipose tissue is more than just a fatty store, though. It is also an active endocrine organ, releasing chemical signals into the bloodstream to signal to the brain and the rest of the body about its current energy state, so the body can adjust its function and behaviour to meet its current needs. Obesity is more than simply being overweight, however. There are major imbalances that occur within the body prior to, and as a result which make this such an important disease in the modern world. During obesity, tissues become desensitised to chemical signals which would usually signal to them to decrease food intake, or to increase energy expenditure. Examples of this are the decreased effect of leptin in the feeding-control-centres of the hypothalamus, leptin on skeletal muscle and adiponectin on the brain and skeletal muscle. These are signals which would ordinarily decrease energy intake by modifying behaviour and metabolism, thus keeping weight down. But in obesity, they become dysregulated. As a result, cells become more prone to store energy as fat, and feeding centres in the brain become impaired. Furthermore, obesity is linked to type II diabetes. The excess expansion of adipocytes from all their fat uptake can stimulate inflammatory immune cells (via hypoxia-induced mechanisms) which can damage the insulin secreting cells of the pancreas. The excess fatty acids present in circulation can also act to inhibit the glucose sensing mechanisms in these insulin secreting cells. This leads to dysregulation of glucose homeostasis, and what we consider to be Diabetes mellitus, aka Type II Diabetes. Adipose as an endocrine organ, and some of its roles in hormonal signalling The underlying biochemistry of obesity shows that there may be certain genetic predispositions which make it more likely for somebody to enter an obese state. Some may shrug this aside as saying ‘You’re just giving fat people an excuse’. But that isn’t the point. Such sentiments merely reflect the negative social stigma attached to obesity, which leads to the psychological impact of obesity on the patient. These social pressures make obesity more than just a disorder of energy regulation, but a disorder which affects psychological state. Predispositions may occur via polymorphism of the structure of hormone-responsive receptors which mediate energy regulating behaviour or metabolism, through variation in horomone secretion patterns, the number of adipocytes one has, amongst many others. Likewise, it’s progression into diabetes relies on multiple imbalances and factors which just complicate the whole diabetes/obesity picture somewhat.









Figure 1. The effects of ghrelin on the CNS, and subsequent glucose, lipid and energy metabolism.

(A) Ghrelin is secreted mainly by the stomach, and can (B) have paracrine or endocrine effects on GI motility or (C) circulate in the blood and act on CNS growth hormone secretagogue receptors (GHS-Rs) inside and outside the blood–brain barrier. Known target areas in the CNS include the hypothalamus, the ventral tegmentum and nucleus accumbens, the hippocampus and GHS-R populations in the brainstem area. The actions of ghrelin in the CNS contribute (D) to the control of food intake and (E) co-regulate tissue-specific cellular pathways in the periphery, thereby governing glucose, lipid and energy metabolism. Control of peripheral metabolism by ghrelin and the CNS is mediated by the autonomic nervous system as well as the hypothalamic–pituitary endocrine axes. Apart from in the stomach, ghrelin is produced in a variety of peripheral tissues, although to a very low extent. (F) Paracrine ghrelin secretion from pancreatic cells might, however, be of importance for the inhibition of insulin secretion from beta cells as well as for beta-cell viability. Abbreviations: BAT, brown adipose tissue; CNS, central nervous system; GI, gastrointestinal; WAT, white adipose tissue.


 

 



Disclaimer: All of the material is not intended to replace the attention or advice of a physician or other qualified healthcare professional. The protocols presented here are one opinion of an integrated approach to investigation and metabolic support with conventional medical approaches to numerous conditions. They should always undertaken with the supervision of physician of other qualified health professional, and I strongly recommend that you verify every recommendation with the current literature and the rapidly evolving art and science of molecular preventive medicine. The biochemical individuality of each human being is paramount. The necessity of blood, urine, stool and tissue testing is essential to tailoring each program to suit the needs of each person who wishes to embark on this journey from a unique state of unwellness and disease to wellness and health.

 
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